Neurobiological Mechanisms of Post-Stroke Hemiplegia
Post-stroke hemiplegia results from ischemic or hemorrhagic disruption of the primary motor cortex or descending corticospinal pathways, severing voluntary neural drive to contralateral skeletal muscles. Patients frequently present with initial flaccidity followed by velocity-dependent hypertonia, clonus, and stereotypic mass synergies.
Restoring functional movement requires structured, repetitive motor relearning that drives synaptic synaptogenesis and perilesional cortical reorganization. Bidaya delivers high-intensity, task-specific motor practice directly within the patient's domestic setting.
